Koska palstan kretardeilla menee suuri osa päivästä murehtien sitä mitä kaksi aikuista ihmistä tekee, lienee aika puhua tästäkin asiasta. Itse en käsitä sitä miksi joku jaksaa viettää päivänsä murehtien toisten (seksi)elämää joka ei ketään satuta, eikä ole laitonta, mutta tapansa kullakin. Luultavinta tosin on, että nämä jotka jaksavat jatkuvasti homouden synnillisyydestä jauhaa ovat itse kaapissa ja kateellisia, mutta katsotaanpas mitä tiede sanoo homoseksuaalisuudesta.
There are, however, a large number of studies indicating that prenatal factors have an important influence on this critical feature of human sexuality.
First, multiple sexually differentiated behavioral, physiological, or even morphological traits are significantly different in homosexual and heterosexual populations. Because some of these traits are known to be organized by prenatal steroids, including testosterone, these differences suggest that homosexual subjects were, on average, exposed to atypical endocrine conditions during development. Second, clinical conditions associated with significant endocrine changes during embryonic life often result in an increased incidence of homosexuality.
Several brain structures have also been shown to be different between homosexual and heterosexual subjects, and in this case, studies focused almost exclusively on males. The first of these differences concerns the suprachiasmatic nucleus, the central clock of the organism, that was shown to be significantly larger in gay men than in heterosexual subjects
The size of the anterior commissure, measured in the midsagital plane, was reported to be larger in gay men than in heterosexual control subjects (43). This difference is more interesting for the purpose of the present discussion, because the size of this commissure is known to be larger in women than in men.
Subsequent studies showed that INAH3 is significantly smaller in homosexual men than heterosexual men, so that its size is essentially equal to what is observed in women
A potential implication of the embryonic hormonal environment in the control of sexual orientation is also supported by studies of various clinical disorders that affect the endocrine system during fetal life. In some cases, these early endocrine disruptions lead to a complete sex reversal, so that, postnatally, subjects are raised assuming a sex (gender) that is opposite to their genetic sex. For example XY subjects with complete androgen insensitivity syndrome are born with female genitalia and are typically raised as girls at least until puberty, when the absence of menstruation leads to medical examination and diagnosis.
There is thus substantial evidence suggesting that sexual orientation, and homosexuality in particular, is influenced before birth by a set of biological mechanisms
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3138231/
Eli homoseksuaalisuus ON SYNYNNÄISTÄ ja se johtuu sikiöajan hormonaalisista seikoista jotka vaikuttavat aivoihin ja niiden fyysiseen kehitykseen. Tämä siis on kliinisten tutkimusten tulos.
Jos haluamme kreationistisen vastauksen tuloksena on se, että kaikki mitä harhainen molesteri horisee on totuus ja lakia ja jos ei noudata sitä on "paha" joka tulee kiduttaa hengiltä.
There are, however, a large number of studies indicating that prenatal factors have an important influence on this critical feature of human sexuality.
First, multiple sexually differentiated behavioral, physiological, or even morphological traits are significantly different in homosexual and heterosexual populations. Because some of these traits are known to be organized by prenatal steroids, including testosterone, these differences suggest that homosexual subjects were, on average, exposed to atypical endocrine conditions during development. Second, clinical conditions associated with significant endocrine changes during embryonic life often result in an increased incidence of homosexuality.
Several brain structures have also been shown to be different between homosexual and heterosexual subjects, and in this case, studies focused almost exclusively on males. The first of these differences concerns the suprachiasmatic nucleus, the central clock of the organism, that was shown to be significantly larger in gay men than in heterosexual subjects
The size of the anterior commissure, measured in the midsagital plane, was reported to be larger in gay men than in heterosexual control subjects (43). This difference is more interesting for the purpose of the present discussion, because the size of this commissure is known to be larger in women than in men.
Subsequent studies showed that INAH3 is significantly smaller in homosexual men than heterosexual men, so that its size is essentially equal to what is observed in women
A potential implication of the embryonic hormonal environment in the control of sexual orientation is also supported by studies of various clinical disorders that affect the endocrine system during fetal life. In some cases, these early endocrine disruptions lead to a complete sex reversal, so that, postnatally, subjects are raised assuming a sex (gender) that is opposite to their genetic sex. For example XY subjects with complete androgen insensitivity syndrome are born with female genitalia and are typically raised as girls at least until puberty, when the absence of menstruation leads to medical examination and diagnosis.
There is thus substantial evidence suggesting that sexual orientation, and homosexuality in particular, is influenced before birth by a set of biological mechanisms
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3138231/
Eli homoseksuaalisuus ON SYNYNNÄISTÄ ja se johtuu sikiöajan hormonaalisista seikoista jotka vaikuttavat aivoihin ja niiden fyysiseen kehitykseen. Tämä siis on kliinisten tutkimusten tulos.
Jos haluamme kreationistisen vastauksen tuloksena on se, että kaikki mitä harhainen molesteri horisee on totuus ja lakia ja jos ei noudata sitä on "paha" joka tulee kiduttaa hengiltä.
Vastaukset
Vanhimmat
Ei vielä vastauksia
Aloittaja odottaa vastauksia. Kerro oma näkemyksesi ensimmäisenä.