The underlying genetic mechanism of evolution is random mutation, and specifically mutation that is beneficial to life. Biology textbooks in theory present positive and negative mutations to students as though these were commonplace and roughly equal in number. However, these books fail to inform students that unequivocally positive mutations are unknown to genetics, since they have never been observed (or are so rare as to be irrelevant).
The biology textbooks in other chapters teach that most mutations are pathologic, or disease-causing, but they don't apply that information to evolution. The worst diseases doctors treat today are caused by genetic mutations. Nearly 4,000 diseases are caused by mutations in DNA.4 "The human genome contains a complete set of instructions for the production of a human being…. Genome research has already exposed errors |mutations| in these instructions that lead to heart disease, cancer, and neurological degeneration."5 These diseases are crippling, often fatal, and many of the affected pre-born infants are aborted spontaneously, i.e., they are so badly damaged they can't even survive gestation. However, the biology textbooks, when discussing mutation in evolution, only discuss the very rare "positive" mutation, like sickle cell anemia. The fact of some 4,000 devastating genetic diseases is suppressed from publication
The biology textbooks in other chapters teach that most mutations are pathologic, or disease-causing, but they don't apply that information to evolution. The worst diseases doctors treat today are caused by genetic mutations. Nearly 4,000 diseases are caused by mutations in DNA.4 "The human genome contains a complete set of instructions for the production of a human being…. Genome research has already exposed errors |mutations| in these instructions that lead to heart disease, cancer, and neurological degeneration."5 These diseases are crippling, often fatal, and many of the affected pre-born infants are aborted spontaneously, i.e., they are so badly damaged they can't even survive gestation. However, the biology textbooks, when discussing mutation in evolution, only discuss the very rare "positive" mutation, like sickle cell anemia. The fact of some 4,000 devastating genetic diseases is suppressed from publication
Vastaukset 16
Vanhimmat
Höpsis, eihän kukaan salaa perinnöllisiä sairauksia ja mutaatioiden osuutta niihin, päinvastoin niistä saadaan ja annetaan jatkuvasti lisää tietoa kiinnostuneille.
However, these books fail to inform students that unequivocally positive mutations are unknown to genetics, since they have never been observed (or are so rare as to be irrelevant).
"However, these books fail to inform students that unequivocally positive mutations are unknown to genetics, since they have never been observed (or are so rare as to be irrelevant)."
LOL. Ja emävalhekin vielä. Hyödyllisiä mutaatioita on hvaittu runsaasti ja esim. laboratoriokokeissa ne ovat rutiinia.
Evokki taas valehtelee. Älköön kukaan kuunnelko häntä.
Avaaja ainakin valehtelee salatessaan lainaamansa tekstin alkuperän. Vaikka se voi näöyttää tieteelliseltä tekstiltä se on ilmiselvää krationistivalhetta.
"Evokki taas valehtelee. Älköön kukaan kuunnelko häntä.
Tuossa sinulle muutamia aidosti hyödyllisiä mutaatioita:
http://en.wikipedia.org/wiki/ApoA-1_Milano
http://en.wikipedia.org/wiki/E._coli_long-term_evolution_experimentResults
Growth in cell size of bacteria in the Lenski experiment
"In the early years of the experiment, several common evolutionary developments were shared by the populations. The mean fitness of each population, as measured against the ancestor strain, increased, rapidly at first, but leveled off after close to 20,000 generations (at which point they grew about 70% faster than the ancestor strain). All populations evolved larger cell volumes and lower maximum population densities, and all became specialized for living on glucose (with declines in fitness relative to the ancestor strain when grown in dissimilar nutrients). Of the 12 populations, four developed defects in their ability to repair DNA, greatly increasing the rate of additional mutations in those strains. Although the bacteria in each population are thought to have generated hundreds of millions of mutations over the first 20,000 generations, Lenski has estimated that within this time frame, only 10 to 20 beneficial mutations achieved fixation in each population, with fewer than 100 total point mutations (including neutral mutations) reaching fixation in each population.[3]"
Miksi sinua pitäisi kuunnella, kun jäit taas kerran valheesta kiinn?
Uskonnonpilkkaaja, uskonrienaaja sepäse täydessä iskussa. Valitettavasti.
Näytä muokkaamaton video hyödyllisestä mutaatiosta. Vain hölmö uskoo asioita jotka on joku tuntematon kirjoittanut nettiin.
http://www.icr.org/article/3466/
Sieltähän se on.
"sitten voin uskoa"
Etkös sinä usko jo nyt Jeesuksen vedenpäällä kävelyyn?
"Näytä muokkaamaton video hyödyllisestä mutaatiosta."
Heh. Hyödyllisiä mutaatioita ei tutkita videoilla, vaan sillä että miten ne leviävät populaatioon ja muokkaavat sen ominaisuuksia. Toki tieteellisesti kyetään tutkimaan myös aminihappojärjestyksiä, joihin tulleet muutokset ovat tuo mutaatio.
"Vain hölmö uskoo asioita jotka on joku tuntematon kirjoittanut nettiin."
Ja vain hölmö ei usko Wikipedian artikkelia, jossa mainitaan tieteellisen tutkimuksen tulos ja tieteelliset lähdeviitteet.
"Pfizer, after the CETP agent torcetrapib failed in a large human safety trial, decided to exit the cardiovascular market in 2008, though they continue to market Lipitor aggressively."
Haitallinenhan tuo on. Ei edes läpäissyt lääketieteellistä tarkastusta.
"Pfizer, after the CETP agent torcetrapib failed in a large human safety trial, decided to exit the cardiovascular market in 2008, though they continue to market Lipitor aggressively."
Haitallinenhan tuo on. Ei edes läpäissyt lääketieteellistä tarkastusta."
Pfizerin kokeilulääke, ei itse mutaatio:
"Esperion, divested by Pfizer in 2008,[10] is back in business and continue to work on HDL mimetic therapies."
"Clinically known as ETC-216, Pfizer did not move trials forward, probably because the complex protein is very expensive to produce and must be administered intravenously, limiting its application compared to oral medications."
Vain umpihölmö uskoo muokkaamattomaksi väitettyyn videoon.
"Vain hölmö uskoo asioita jotka on joku tuntematon kirjoittanut nettiin."
Tieteellisen tutkimusraportin takana on aina useampikin jonkun tuntema henkilö.
Todista se. Se, että jossain lukee että se on tehty näin ja näin ja tarkastettu näin ja näin ei ole vielä mikään tae siitä että se olisi totta! Tiesitkö että suurin osa tieteellisistä julkaisuista on huijauksia. Moni on kopioinut vanhoja julkaisuja ja julkaissut ne omalla nimellään. Veikkaan Dawkinsia tällaiseksi.
Varmaankin myös uskot puhuvaan käärmeeseen?
Sitäpä en tiedä vieläkään:
"Tiesitkö että suurin osa tieteellisistä julkaisuista on huijauksia."
Sensijaan tiedän, että tuo väitteesi on törkeä vale.